BACKGROUND: Flavobacterium meningosepticum is an uncommon pathogen causing nosocomial pneumonia and meningitis in newborns. It is usually. Elizabethkingia meningoseptica is a gram-negative rod-shaped bacterium widely distributed in . Flavobacterium meningosepticum King, (Approved Lists, ); Chryseobacterium meningosepticum (King, ) Vandamme et al., . Flavobacterium meningosepticum is a gram negative, opportunistic organism which has been implicated in a number of outbreaks of sepsis and meningitis in.

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The baby was assessed to be small for gestational age with postnatal sepsis and patent ductus arteriosus. Associated Data Supplementary Materials. About Us Contact Us. Macromolecule Content Total Structure Weight: Open in a separate window. Eighteen isolates of F meningosepticum were identified from 16 patients. Reducing the use of unnecessary residual devices and invasive procedures may help reduce the incidence of infection. F meningosepticum is an opportunistic pathogen of low virulence and rarely causes serious infections in adults.

Find similar proteins by: Lumbar puncture was done after infusion of platelets and fresh frozen plasma.

Cefotaxime and Amikacin were continued after blood and CSF cultures. Subcommittee on the taxonomy of Flavobacterium and Cytophaga -like bacteria. Flavobacteria Bacteria described in Neonatal meningitis caused by flavobacterium meningosepticum. The bacterium is also a rare cause of nosocomial pneumoniaendocarditispost-operative bacteremiaand meningitis meninbosepticum immunocompromised adults. Neonatal meningitis caused by Flavobacterium meningosepticum type F. At the meeting of the International Committee on Systematics of Prokaryotes, the organization’s subcommittee on the taxonomy of Flavobacterium and Cytophaga-like bacteria named J.


Flavobacterium meningosepticum bacteremia: an analysis of 16 cases.

Initially the organism which was cultured from CSF was suspected to be Pseudomonas and therapy was changed to carbenicillin and tobramycin. It is usually resistant to antimicrobial agents used to treat gram-negative bacilli. Head circumference had increased to 44 cm from Testing with lomefloxacin, ciprofloxacin and ofloxacin yielded Bacteria plated in this way may not show yellow color.

Meningitis caused by Flavobacterium meningosepticum. These two can be distinguished by the indole test or the Pyr testboth of which should be clearly negative for B. Successful treatment of the F. Of them, seven were suspected to have acquired the pathogen from diagnostic or therapeutic procedures.

Research articles conducted on animals, will not be considered for processing or publication in the JPMA. You can also find us on social media:.

International Journal of Systematic and Evolutionary Microbiology.


KingKim et al. There were 10 men and six women, with a mean age of American Journal of Clinical Pathology. Colonies are very pale yellow and may not be easily evident at 24 hours. Exchange transfusion was performed and the baby was mechanically ventilated.


Flavobacterium meningosepticum.

Nosocomial meningitis of the newborn caused by a flavobacterium. Ventricular CSF cultured at this time was sterile.

Funding is constantly needed for new projects and to update and refurbish existing facilities. This bacterium is usually multiresistant to antibiotics typically prescribed for treating gram-negative bacterial flavobactsrium, including extended-spectrum beta-lactam agents due to production by most strains of two betalactamases: Neonatal meningitis caused by new serotype of Flavobacterium meningo-septicum.


The organism was resistant to penicillins, cephalosporins, aztreonam, imipenem, aminoglycosides and macrolides. Bacteremia occurred in these patients within a mean period of 2. While the pathogen often results in high mortality and serious sequelae in newborns, it is also found to cause to disease in adults. Although the organism was resistant to rifampicin and ceftazidime on disc diffusion testing, ceftizoxime was not readily available and an interim therapy was initiated with these drugs.

The results confirm the suitability of the bacterial enzyme as a model to study the consequences of mutations in aspartylglycosaminuria patients. The active site of Meningoseoticum glycosylasparaginase is in an open conformation when compared with the human structure.